34 Results for : infarct

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    Brain and Heart Infarct II ab 117.49 EURO Softcover reprint of the original 1st ed. 1979
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    The Cerebral Infarct ab 90.99 EURO Pathology Pathogenesis and Computed Tomography. Softcover reprint of the original 1st ed. 1985
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    Diffusion-weighted MR imaging is widely accepted as a means to identify stroke, thus enabling rapid and effective treatment. Over the past four years, these expert authors have presented over 30 exhibits and scientific reports on diffusion-weighted imaging at the RSNA and the American Society of Neuroradiology (ASNR), and more than 10 of these presentations have been recognized by specific awards. Diffusion-Weighted MR Imaging of the Brain's chapters range from basic principles to interpretation of diffusion-weighted MR imaging and specific disease. This is a valuable reference for radiologists, neurologists, neurosurgeons as well as residents, fellows, radiology technologists. TOC:Principles of diffusion-weighted imaging.- Normal brain and artifacts.- Brain edema: Classification. Cytotoxic edema. Edema of neuron and glial cell. Intramyelinic edema. Axonal edema. Vasogenic edema.- Infarction: Arterial infarction. Venous infarction.- Dementia: Alzheimer's. Multi-infarct dementia. Other dementias. HIV dementia.- Hemorrhage.- Vasculitis/Vasculopathy: Behcet. Systemic lupus erythematosus. Posterior reversal encephalopathy syndrome. Drug induced.- Epilepsy: Postictal encephalopathy. Status epilepticus.- Demyelinating and degenerative diseases: ADEM. Multiple sclerosis. Creutzfeldt-Jakob disease.- Toxic and metabolic diseases: Drug-induced encephalopathy. Mitochondrial encephalopathy. Phenylketonuria. Osmotic myelinolysis. Marchiafava-Bignami disease.- Infectious diseases: Brain abscess. Extra-axial abscess. Encephalitis. Septic emboli.- Trauma: Diffuse axonal injury. Brain contusion.- Brain neoplasm: Epidermoid. Meningioma. Lymphoma. GBM. Metastasis. PNET.- Pediatrics.
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    Arterial hypertension, coronary heart disease and heart fail ure are the commonest cardiovascular conditions to present in clinical practice. Over the past few years it has become in creasingly clear that they are closely and causally interrelated and that their relationship can have a significant bearing on prognosis. Epidemiological studies have shown that arterial hypertension is one of the most important risk factors for de veloping heart failure. Only one in four patients with hyper tension is adequately managed, and in 50% of cases, the hypertension has not been recognised or treated. Patients with pre-existing hypertension who go on to suffer an acute myocardial infarction have usually not previously had typi cal angina symptoms, the infarct territory is larger, life threatening arrhythmias are commoner and hence in-hospi tal mortality and long-term prognosis are markedly worse. The presence of raised blood pressure in the post-infarct phase doubles the risk of manifest heart failure. The close relationship between hypertension, coronary heart disease and heart failure makes the choice of therapeu tic strategy particularly important. Agents and classes of agents that have prognostic value in all three conditions should be considered first, as synergy might result in addi tional benefits. In such patients, this sort of therapeutic deci sion-making might have further advantages. The use of these agents may prevent complications which are not yet clinically obvious (such as heart failure).
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    Acute Myocardial Infarction is one of the leading causes of cardiovascular deaths. Left ventricular thrombus is a frequent and potentially dangerous complication of acute myocardial infarction, and is associated with increased risk of systemic embolization and higher mortality rates after acute myocardial infarction. Mural thrombosis with embolism typically occurs in the setting of a large (especially anterior) ST segment elevation acute MI. Thus, in patients with anterior ST segment elevation acute MI and in other high-risk patients, echocardiography should be performed during hospitalization to detect LV thrombus. This work is designed to highlight the magnitude of left ventricular thrombus in patients suffering from acute anterior myocardial infarction. The results of this study can be used to guide future recommendations for importance of diagnosis of this complication and guide treatment strategies in the post infarct patients, because significant frequency of left ventricular thrombus warrant early diagnosis and management of this prognostically life threatening, yet silent complication of myocardial infarction.
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    The concept of dementia has itself been the subject of increasing refinement and precision in definition and diagnosis. Two important sub-types have been identified: Alzheimer's disease and multiple-infarct dementia. Alzheimer's disease or Senile Dementia of the Alzheimer Type (SDA T), arises out of changes in the brain which are as yet poorly understood and identifiable with certainty only at post mortem examination. This type of dementia has been named after Alois Alzheimer, who first identified these changes, in the earliest years of this century. Alzheimer's is the commonest type of identified dementia. The second commonest type is multi-infarct dementia (MID), which follows a stroke or strokes affecting the relevant part of the brain. Like SDA T, it can be diagnosed with certainty only after death, although Jorm (1987, Ch. 8) reviews progress in using various types of tests which can suggest, if not confirm, its presence during the sufferer's lifetime. As will be shown in Chapter 2, some populations appear to reverse the general picture and exhibit more MID than SDAT. Many dementia sufferers cannot, even after a post-mortem examination, be neatly categorized as one or the other of these sub-types. A considerable uncertain 'grey area' of dementia remains at present very poorly understood. Some elderly people develop dementia as a side-effect of known physical disease. Some of these conditions are curable. Care of the demented person has traditionally taken place in the community: hospitalization is a fairly recent innovation.
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    1.1. Definition of Terms-Thrombosis, Thromboembolic Disease, Atherosclerosis, and Blood Clotting The terms heart attack or myocardial infarction are more commonly used than thrombosis. The infarct-muscle destruction is simply the end result and thrombosis is the real cause of the heart attack. Thrombosis may be defined as the process of formation of a coalescent or agglutinated solid mass of blood components in the blood stream. Thrombi formed in either arteries or veins often cause occlusion in the vascular system and prevent blood flow. Obstruc to the blood vessel usually occurs at the site where the thrombi deposit. tion Furthermore, thrombi may break loose, travel through the circulating blood stream, and cause obstruction at some distal point of narrowing elsewhere. The mass or thrombus that moves is referred to as an 'embolus.' The two phenomena are lumped together under the term thromboembolic disease. Thrombosis that reduces blood supply to the heart is the primary factor in heart attacks.
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    The major aspect of treatment of STEMI is reperfusion of the infarct related artery. Either thrombolytic therapy or primary angioplasty. Thrombolytic therapy is the older and easy to perform, where as primary angioplasty is relatively new technique and lots of skill and catheter laboratory setup is needed. We compare the short term outcome between Primary PCI (Group-I) and intravenous Streptokinase (Group-II) as reperfusion therapy for the management of acute STEMI. Pre and post procedural (1) ECG changes. (2) Improvement of TIMI flow. (3) Improvement of LV function (LVEF) and (4) Complications were compared. Study showed more improvement of ECG changes, TIMI flow and LVEF in group I patients. In complications, Primary PCI group had less MACE but no significant difference in vascular complications between two groups. This study conclude that the patient received Primary PCI has better in-hospital outcome in comparison to patient received intravenous Streptokinase for reperfusion therapy in STEMI. This book should be of interest to Interventional Cardiologists, Clinical Cardiologists,nurses of CCU & ICU and Physicians who are interested in the management of Acute Cardiac Care.
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    Are there universal laws governing the persistence of weather, and is it possible to predict climate transitions as generated by natural or man-made perturbations? How can one quantify the roller-coaster dynamics of stock markets and anticipate mega-crashes? Can we diagnose the health condition of patients from heartbeat time-series analysis, which may even form the basis for infarct prevention? This book tackles these questions by applying advanced methods from statistical physics and related fields to all types of non-linear dynamics prone to disaster. The transdisciplinary analysis is organized in some dozen review articles written by world-class scientists.
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    The electrocardiogram (ECG) remains the most accessible and inexpensive diagnostic tool to evaluate the patient presenting with symptoms suggestive of acute myocardial ischemia. It plays a crucial role in decision making about the aggressiveness of therapy especially in relation to reperfusion therapy, because such therapy has resulted in a considerable reduction in mortality from acute myocardial infarction. Several factors play a role in the amount of myocardial tissue that can be salvaged by reperfusion therapy, such as the time interval between onset of coronary occlusion and reperfusion, site and size of the jeopardized area, type of reperfusion attempt (thrombolytic agent or an intracoronary catheter intervention), presence or absence of risk factors for thrombolytic agents, etc. Most important in decision making on reperfusion therapy and the type of intervention is to look for markers indicating a higher mortality rate from myocardial infarction. The ECG is a reliable, inexpensive, non-invasive instrument to obtain that information. Recently it has become clear that both in anterior and inferior myocardial infarction, the ECG frequently allows not only to identify the infarct related coronary artery, but also the site of occlusion in that artery and therefore the size of the jeopardized area. Obviously, the more proximal the occlusion, the larger the area at risk and the more aggressive the reperfusion attempt.
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